★ Is Cancer's Secret Ally Sitting on Your Plate?
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Is Cancer's Secret Ally Sitting on Your Plate?
New findings suggest this overlooked nutrient may quietly fuel tumor growth, disrupt your hormones, and help cancer cells slip past your immune system undetected for years.
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Terrible article that should not have been sent. There is no definition of "A HIGH FAT DIET". This is neither convincing nor actionable. We have long known that abdominal fat is inflammatory and activates platelets which increase cancer of all kinds. Many high fat diets do not make people fat and are healthy. Which fats is the key question, these authors seem oblivious to the central issue.
Lung cancer is currently the leading cause of cancer death worldwide; it is often diagnosed at an advanced stage and bears poor prognosis. It has been shown that diet is an important environmental factor that contributes to the risk and mortality of several types of cancers. Intake of ω-3 and ω-6 PUFAs plays an important role in cancer risk and progression. Current Western populations have high consumption of ω-6 PUFAs with a ratio of ω-6/ω-3 PUFAs at 15:1 to 16.7:1 This high consumption of ω-6 PUFAs is related to increased cancer risk and progression. However, whether a diet rich in ω-6 PUFAs can contribute to tumor aggressiveness has not been well investigated. We used a murine model of pulmonary squamous cell carcinoma to study the aggressiveness of tumors in mice fed with a diet rich in ω-6 PUFAs and its relationship with oxylipins. Our results shown that the mice fed a diet rich in ω-6 showed a marked increase in proliferation, angiogenesis and pro-inflammatory markers and decreased expression of pro-apoptotic proteins in their tumors. Oxylipin profiling revealed an upregulation of various pro-tumoral oxylipins including PGs, HETEs, DiHETrEs and HODEs. These results demonstrate for the first time that high intake of ω-6 PUFAs in the diet enhances the malignancy of tumor cells by histological changes on tumor dedifferentiation and increases cell proliferation, angiogenesis, pro-inflammatory oxylipins and molecular aggressiveness targets such as NF-κB p65, YY1, COX-2 and TGF-β.
https://www.mdpi.com/1422-0067/23/11/6179