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Just steve
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Some sources report Live Fermented Kim Chi binds, sucks up microplastics. Kim Chi on the regular is excellent for Gut Health. So whether or not the Chi binds the plastic up and removes it directly out of the Gut, or if it enhances the health of the Gut Lining to prevent the plastic from invading the body that way, or if the Chi does both (Seems likely both,) gives it a King status as a side dish. Also along the same lines an experiment using the left over sauerkraut juice on fields rejuvenates the probiotics in the soil and/or in turn 'removes' the glyphosate. Might kraut too remove the glyphosate from our Gut? Regardless this suggests the power of living fermented foods building a solid foundation of Health for our bodies to stand on.

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Guillermou
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A grim picture of environmental pollution from microplastics and nanoplastics. The problem is that once internalized orally (through ingestion, inhalation, or dermal contact), microplastics can remain in tissues indefinitely due to their resistance to biodegradation. Their small size allows them to cross epithelial barriers and thus accumulate in organs such as the liver, kidneys, intestines, and possibly the brain. This accumulation of microplastics is a major concern when considering long-term exposure scenarios with chronic low doses, which are more representative of real-world situations. Unlike acute toxic agents, prolonged exposure to low doses may produce very few clinical signs; however, it can induce chronic oxidative stress, inflammation, immune dysregulation, and genotoxicity—all indicators of carcinogenesis. Ultimately, chronic cellular stress can lead to DNA damage, obstruct DNA repair mechanisms, and interfere with signaling pathways, thus affecting the initiation and progression of carcinogenesis. The cumulative nature of prolonged exposure to low doses of microplastics necessitates mechanistic studies and regulatory interest, as even the smallest amount of microplastic exposure can significantly impact human health over decades.

Figure 2 of the first link illustrates the pathways of microplastic accumulation (a) and potential deposition sites (b) in the human body. The diagram highlights the main routes of exposure: ingestion and dermal contact. Deposition sites include the gastrointestinal tract, lungs, and systemic circulation, with potential bioaccumulation in organs such as the liver, kidneys, and lymphatic system.

Microplastics are dispersed throughout our environment from factories, cities, synthetic clothing, and personal products. These tiny plastic fragments move through living and non-living systems, creating complex pollution patterns that threaten ecological balance and human health. Understanding their diverse sources and interactions is crucial for developing effective management strategies. Adapted with permission from the American Chemical Society. Copyright 2024. (b) The schematic representation shows the sources, distribution, and dynamic behavior of microplastics within atmospheric systems. See Fig. 3, first link.

Due to their resistance and difficulty in degrading, microplastics persist for a long time in many places. The degradation of conventional plastics in nature is very limited. Most synthetic materials resist degradation by microorganisms due to their heavy molecular structure and the absence of functional groups that microorganisms can easily recognize. Since microplastics can carry additional contaminants, the problem is significantly more complex. Furthermore, viruses and heavy metals can adhere to the surfaces of microplastics, which could increase their availability and mobility in the environment. This effect not only makes these contaminants more prevalent but also raises concerns about whether microplastics could be an entry point for hazardous substances that accumulate in living organisms.

Studies specify that microplastics can cross barriers such as the blood-brain barrier and the placental barrier, possibly affecting fetal development and brain function.

The infiltration of microplastics into tissues is influenced by their physical characteristics, such as size, shape, and chemical composition. While the long-term impact of microplastic accumulation in human tissues is still being studied, preliminary research indicates that exposure could lead to inflammation, tissue damage, and potential associations with diseases such as cancer. Furthermore, ongoing studies are exploring the relationships between microplastics, the human microbiome, and the immune system, raising concerns about the potential disruption of normal bodily functions. Results demonstrate that microplastics can exacerbate tumor cell development by affecting important mechanisms such as oxidative stress, immunosuppression, and the modulation of critical signaling pathways. Research on hematological cancers highlights the presence of microplastics in human blood, suggesting their potential systemic distribution and impact. The ability of microplastics to cross the blood-brain barrier raises concerns about brain cancer, where they could induce neurotoxicity. Similarly, microplastics contribute to colorectal cancer by causing intestinal inflammation and alterations in the gut microbiota. Microplastic (MP) inhalation is linked to lung cancer due to chronic inflammation and oxidative stress.

In liver cancer, MP induce liver toxicity and promote carcinogenesis. Breast and cervical cancers are associated with the endocrine-disrupting properties of MP, leading to increased cell proliferation and migration. There is an urgent need for further research to elucidate the mechanisms by which MP contribute to cancer and to inform public health strategies and regulatory policies aimed at mitigating the risks of microplastic exposure. Possible mechanisms have been described by which microplastics can disrupt the mucus layer, thereby reducing its protective effect and increasing the likelihood of colorectal cancer. Furthermore, microplastics also triggered gut microbiota dysbiosis, which significantly contributed to resistance to immunotherapy in colorectal cancer. Microplastics play a significant role in colorectal cancer resistance to immunotherapy by disrupting the tumor's immune microenvironment and represent a novel target in colorectal cancer immunotherapy, especially amidst increasing microplastic pollution. Individuals born after the plastic boom of the 1950s have been continuously exposed to microplastics from the womb, potentially predisposing them to carcinogenic pathways later in life at an early age.

https://pubs.rsc.org/en/content/articlehtml/2025/em/d5em00232j (2025)

https://www.sciencedirect.com/science/article/abs/pii/S0098299724000955 (2025)

https://www.dl.begellhouse.com/journals/0ff459a57a4c08d0,2398aadc50463bfa,73884b634ac553a0.html (2025)

https://www.mdpi.com/2072-6694/15/13/3323 (2025)

https://journals.lww.com/eurjcancerprev/abstract/9900/microplastics_and_cancer.248.aspx (2025)

https://www.sciencedirect.com/science/article/pii/S0304389426003109 (2026)

https://www.taylorfrancis.com/chapters/edit/10.1201/9781003644309-11/plastic-pollution-cancer-niketha-manoj-shabina-ashraf (2026)

https://link.springer.com/article/10.1186/s12943-026-02588-8 (2026)

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https://www.sciencedirect.com/science/article/pii/S0304389426008757 (2026)

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