Mammograms are said to reduce the risk of death by 20%, but research demonstrates this may not be true and that it may actually do more harm than good.
Most xenobiotics are metabolized or/and bioaccumulate and biomagnify in our tissues and cells, including breast tissues, so the metabolism of xenobiotics plays an important role in the initiation and progression of breast cancer (BC). . Existing evidence on breast cancer risk (BCR) suggests that foodborne chemical carcinogens, environmental pollution, ionizing radiation, and socioeconomic status are closely related to breast carcinogenesis. At the level of biological pathways, most xenobiotics interact with endocrine signaling, adipogenesis, angiogenesis, DNA repair, inflammatory response, IGF-1 and NF-κB signaling, PI3K/Akt signaling, fatty acid metabolism and glycolysis, the p53 pathway, xenobiotic metabolism and other cancer-related pathways.
Breast cancer is higher for women who work at night, since exposure to light at night suppresses the secretion of melatonin and leads to cancer. Exposure to xenoestrogen (present in plastic packaging, pesticides, etc.), parabens (present in preservatives and cosmetics),
Alkylphenols and phytoestrogens are really harmful and can cause BC. In addition, excessive alcohol consumption, diabetes, obesity and late onset of menopause are also responsible for breast cancer.
During puberty, a woman's breasts are vulnerable to environmental damage. Early exposure to environmental carcinogens, endocrine disruptors, and unhealthy foods (refined sugar, processed fats, food additives) promotes molecular damage that increases breast cancer risk.
Environmental exposure to chemicals, such as endocrine-disrupting chemicals, is increasingly recognized as contributing to the development of breast cancer.
The use of cosmetics may be associated with undesirable effects due to the presence of certain chemical substances. Among 50 random facial makeup cosmetics, the following substances were identified as potential carcinogens: parabens, ethoxylated compounds, formaldehyde donors, and ethanolamine and their derivatives.
Also consider the danger of deodorants with parabens and/or aluminum. In general, topical application of personal care products contain estrogen-mimicking parabens, with even more risk from aluminum in antiperspirants.
Aluminum chloride, the active ingredient in antiperspirants, acts similar to the way oncogenes act to provide molecular transformations in cancer cells. Like parabens, aluminum salts also mimic estrogen and bioaccumulate in breast tissue, which may increase the risk of breast cancer.
Parabens are a group of EDCs commonly found in personal care products, foods, and pharmaceuticals. Systemic exposure to parabens has been confirmed by the ubiquitous detection of parabens in human blood and urine samples. Although evidence from in vivo and epidemiological studies linking paraben exposure to breast cancer is limited, current evidence suggests that parabens may negatively interfere with some endocrine and intracrine targets relevant to breast carcinogenesis.
Most xenobiotics are metabolized or/and bioaccumulate and biomagnify in our tissues and cells, including breast tissues, so the metabolism of xenobiotics plays an important role in the initiation and progression of breast cancer (BC). . Existing evidence on breast cancer risk (BCR) suggests that foodborne chemical carcinogens, environmental pollution, ionizing radiation, and socioeconomic status are closely related to breast carcinogenesis. At the level of biological pathways, most xenobiotics interact with endocrine signaling, adipogenesis, angiogenesis, DNA repair, inflammatory response, IGF-1 and NF-κB signaling, PI3K/Akt signaling, fatty acid metabolism and glycolysis, the p53 pathway, xenobiotic metabolism and other cancer-related pathways.
Breast cancer is higher for women who work at night, since exposure to light at night suppresses the secretion of melatonin and leads to cancer. Exposure to xenoestrogen (present in plastic packaging, pesticides, etc.), parabens (present in preservatives and cosmetics),
Alkylphenols and phytoestrogens are really harmful and can cause BC. In addition, excessive alcohol consumption, diabetes, obesity and late onset of menopause are also responsible for breast cancer.
During puberty, a woman's breasts are vulnerable to environmental damage. Early exposure to environmental carcinogens, endocrine disruptors, and unhealthy foods (refined sugar, processed fats, food additives) promotes molecular damage that increases breast cancer risk.
Environmental exposure to chemicals, such as endocrine-disrupting chemicals, is increasingly recognized as contributing to the development of breast cancer.
The use of cosmetics may be associated with undesirable effects due to the presence of certain chemical substances. Among 50 random facial makeup cosmetics, the following substances were identified as potential carcinogens: parabens, ethoxylated compounds, formaldehyde donors, and ethanolamine and their derivatives.
Also consider the danger of deodorants with parabens and/or aluminum. In general, topical application of personal care products contain estrogen-mimicking parabens, with even more risk from aluminum in antiperspirants.
Aluminum chloride, the active ingredient in antiperspirants, acts similar to the way oncogenes act to provide molecular transformations in cancer cells. Like parabens, aluminum salts also mimic estrogen and bioaccumulate in breast tissue, which may increase the risk of breast cancer.
Parabens are a group of EDCs commonly found in personal care products, foods, and pharmaceuticals. Systemic exposure to parabens has been confirmed by the ubiquitous detection of parabens in human blood and urine samples. Although evidence from in vivo and epidemiological studies linking paraben exposure to breast cancer is limited, current evidence suggests that parabens may negatively interfere with some endocrine and intracrine targets relevant to breast carcinogenesis.
https://www.mdpi.com/1660-4601/19/3/1873 (2022).--
https://medcraveonline.com/JCPCR/JCPCR-13-00492.pdf (2022).--
https://www.mdpi.com/1660-4601/17/2/493 (2020).--
https://www.sciencedirect.com/science/article/abs/pii/B9780323996846000112 (2023).-
https://www.mdpi.com/1660-4601/20/6/4780 (2023).--
- https://www.mdpi.com/2075-1729/14/3/402 (2024).--